| 1 |
Podocyte RNF166 deficiency alleviates diabetic nephropathy by mitigating mitochondria impairment and apoptosis via regulation of CYLD signal
Ma HB, Dong YJ, Wang S, Sun K, Liu YJ, Li MM
Biochemical and Biophysical Research Communications, 545, 46, 2021 |
| 2 |
Ablation of lncRNA MIAT mitigates high glucose-stimulated inflammation and apoptosis of podocyte via miR-130a-3p/TLR4 signaling axis
Zhang M, Zhao SM, Xu C, Shen Y, Huang J, Shen S, Li YC, Chen XM
Biochemical and Biophysical Research Communications, 533(3), 429, 2020 |
| 3 |
DUSP26 regulates podocyte oxidative stress and fibrosis in a mouse model with diabetic nephropathy through the mediation of ROS
Huang F, Sheng XX, Zhang HJ
Biochemical and Biophysical Research Communications, 515(3), 410, 2019 |
| 4 |
Down-regulation of IRAK1 attenuates podocyte apoptosis in diabetic nephropathy through PI3K/Akt signaling pathway
Zhang Y, Chen XF, Yuan L, Zhang YD, Wu JH, Guo NF, Chen X, Liu J
Biochemical and Biophysical Research Communications, 506(3), 529, 2018 |
| 5 |
TMEM16A exacerbates renal injury by activating P38/JNK signaling pathway to promote podocyte apoptosis in diabetic nephropathy mice
Lian H, Cheng Y, Wu XY
Biochemical and Biophysical Research Communications, 487(2), 201, 2017 |
